Ukuqonda uQhubeko olusuka kwizilonda zePrecancerous ukuya kuMhlaza

Umhlaza awuphuhli ngobusuku; kunoko, ukuqala kwayo yinkqubo ngokuthe ngcembe ebandakanya izigaba ezithathu: izilonda ze-precancerous, i-carcinoma in situ (amathumba okuqala), kunye nomhlaza ohlaselayo.

Izilonda ezinomhlaza zisebenza njengesilumkiso sokugqibela somzimba phambi kokuba umhlaza ubonakale ngokupheleleyo, emele imeko elawulekayo nenokubuyiselwa umva. Noko ke, enoba olu tshintsho luhlehla okanye luwohloka luxhomekeke kwizenzo zikabani.

Yintoni izilonda zePrecancerous?

Okokuqala, kubalulekile ukuqaphela ukuba amanxeba angaphambi komhlaza ayingomhlaza; azinayo iiseli zomhlaza. Banokujongwa njengezalamane ezisondeleyo zomhlaza, ezinokuthi ziguquke zibe ngumhlaza phantsi kwempembelelo yexesha elide ye-carcinogens. Ngoko ke, azilingani nomhlaza kwaye akufanele zidibanise.

Ukuvela kwizilonda ezingaphambi komhlaza ukuya kumhlaza yinkqubo ehamba kancinci, ihlala iminyaka eliqela okanye amashumi eminyaka. Eli xesha libekiweyo linika umntu ngamnye ithuba elaneleyo lokungenelela. Izilonda ezinomhlaza zibangelwa yimiba eyahlukeneyo, kuquka ukosuleleka okanye ukudumba okungapheliyo, indlela yokuphila engafanelekanga, kunye nemfuza. Ukuchonga izilonda ze-precancerous akusona siphumo esibi; lithuba lokungenelela kwangexesha, ukunqandwa kwamathumba anobungozi, kunye nokubuyisela umva. Amanyathelo anjengokususwa kotyando, ukuphelisa ukudumba, kunye nokuthintelwa kwezinto ezivuselelayo kunokubuyisela izilonda ezinobungozi kwimeko yesiqhelo.

Ayingawo onke amathumba abonisa izilonda eziqhelekileyo, ezibonwa lula. Izilonda ezixhaphakileyo ze-precancerous ezidibana neklinikhi ziquka:

• Ukuthintela uMhlaza Wesisu: Lumkela i-Chronic Atrophic Gastritis

• Amanqanaba ophuhliso: Isisu sesisu esiqhelekileyo → isifo segastritis esingapheliyo → Igastritis engapheliyo yeatrophic

• Utshintsho lwe-Histological: I-Metaplasia yamathumbu, i-dysplasia

• Isiphumo sokugqibela: Umhlaza wesisu

Nangona i-atrophic gastritis engapheliyo ingaqhubeli phambili kumhlaza wesisu, iimeko ezinganyangwanga okanye ukuvuselela ngokuphindaphindiweyo (njengokusela kakhulu utywala, i-bile reflux, usulelo lwe-Helicobacter pylori, okanye ukusetyenziswa kwexesha elide lamayeza athile) kunokunyusa umngcipheko womhlaza.

Iimpawu zeklinikhi ziquka:

• Isicaphucaphu kunye nokugabha

• Ukuxhatshazwa kwesisu kunye neentlungu

• Ukulahlekelwa ngumdla wokutya

• Ukubhinca

• Ukuthintela uMhlaza oMbala: Sukuyijongela phantsi iAdenomatous Colorectal Polyps

• Amanqanaba okuqhubela phambili kwesifo: Umhlaza wobhobhobhobhosi → ukudumba kwamathumbu → Iipolyp ezingamathanga → ithumba elipolypoid

• Ixesha lenguquko: I-Benign polyps kumhlaza ihlala ithatha iminyaka emi-5 ukuya kweli-15.

Iimpawu ze-adenomatous colorectal polyps:

• Ukunyuka kwamathumbu

• Intlungu zesisu

• Ukuqunjelwa

• Ilindle elinegazi

Ukuthintela uMhlaza Wesibindi: Gcina iliso elisondeleyo kwi-Cirrhosis yesibindi

Amanqanaba okuqhubela phambili: IHepatitis → Ukuqina kwesibindi → Umhlaza wesibindi

Imiba yomngcipheko: Abantu abanembali ye-hepatitis B kunye ne-cirrhosis ehamba nesibindi basengozini enkulu yomhlaza wesibindi.

Iindlela zongenelelo:

• Iimviwo rhoqo: Isibindi B-ultrasound kunye novavanyo lwenqanaba le-alpha-fetoprotein rhoqo kwiinyanga ezi-3-6 kwizigulane ezine-hepatitis B-ehlobene ne-cirrhosis.

• Ukujongwa okusebenzayo kokuphindaphinda kwentsholongwane ye-hepatitis B kunye nonyango olusemgangathweni lwe-antiviral kwizigulana ze-hepatitis B.

• Amanye amanyathelo okuthintela: Ukutshaya nokuyeka utywala, kunye nokuphepha ukusebenza kakhulu.

• Ukuthintela uMhlaza wamabele: Lumka kwi-Atypical Breast Hyperplasia

Inkqubo ngokubanzi: Ibele lesiqhelo → I-hyperplasia engekhoyo → Carcinoma in situ → I-hyperplasia yebele → Hyperplasia → Umhlaza wamabele